Mayo Clinic Health Letter provides reliable, easy-to-understand, health and medical information. The arthritis-related pain in your knees has steadily worsened over the insulin resistance diet pdf years.
Now, walking even a few blocks is almost unbearable, getting out of a chair causes you to wince, and your knees have a dull ache, even in bed. A lifetime of normal wear and tear on ankle joints commonly causes ankle osteoarthritis in older adults. Studies show that synthetic, genetically modified insulin could be to blame for a number of complications in diabetes patients. Journal Articles copyright of original owners, MeSH copyright NLM.
Likewise, in a study of 84,622 type 2 diabetes patients, insulin monotherapy increased risk of major adverse cardiac events by 73. Insulin administration also increased neuropathy by 2. 146-fold, renal complications by 3. 504-fold, and all cause mortality by 2. In fact, survival rate at one year was 85. Whereas the death rate was 8. 63 per 100 patients-year in non insulin-treated diabetics, it was 15.
We also observed a strong gradient of cumulative insulin dispensations and cancer mortality rates in this population. Because interventional studies are scarce, much of the scientific data on this subject matter is derived from epidemiological studies. In one Japanese study, insulin administration led to rapid deterioration of glucose control, decline in C-peptide to levels reflective of insulin deficiency, and onset of type 1 diabetes after a mean duration of 7. Is Genetically Modified Insulin to Blame? 1 diabetes following treatment of insulin-resistant type 2 diabetes, as well as the host of other adverse sequelae associated with insulin use in type 2 diabetics, may be due in part to the genetic recombinant insulin preparations that are the brainchild of the biotech industry.
Changes in primary structure, or sequence of amino acid residues, are translated into changes in geometric secondary structure, interactions between motifs known as super secondary structure, and interactions among protein domains, such as disulfide bonding, known as tertiary structure. Due to the principal that structure governs function, these modifications can result in deleterious functional changes. Because it is unable to assume the native protein structure, GM insulin may not only be unable to perform the life-sustaining functions of endogenous insulin, but it also has the potential to elicit maladaptive immune responses. In many cases, insulin treatment in type 2 diabetes, a disorder of deranged insulin signaling, may represent adding fuel to the fire. No individual should misconstrue this article as medical advice or discontinue any medication without the approval of a licensed physician. The intent, rather, is to encourage early dietary and lifestyle interventions, combined with targeted nutraceuticals, in order to prevent the downward trajectory that results in the prescribing of insulin to treat type 2 diabetes.
Consistent with naturopathic precepts, health care providers should prioritize these lowest risk, least invasive interventions as the standards of care. Type 2 Diabetes Mellitus: A Review of Current Trends. Etiology of type 1 diabetes. The metabolic syndrome—a new worldwide definition.
Mitochondrial pathophysiology and type 2 diabetes mellitus. Pathophysiology of type 2 diabetes and the role of incretin hormones and beta-cell dysfunction. Journal of the American Academy of Physicians, Suppl 3-8. Department of Health and Human Services.